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One study demonstrated that apatinib inhibited the expression of GLS1, activated the GCN2/eIF2/ATF4 pathway, and promoted the expression of SLC1A5, thereby reprogramming Gln metabolism in NSCLC and weakening the response of tumor cells to apatinib (33)

Paradoxically, mice lacking gamma-glutamyl transpeptidase (GGT)an enzyme essential for GSH degradationdeveloped milder fibrosis following BLM exposure [126]